Richard N. Fogoros(auth.)'s Antiarrhythmic Drugs: A Practical Guide, Second Edition PDF

By Richard N. Fogoros(auth.)

ISBN-10: 0470692529

ISBN-13: 9780470692523

ISBN-10: 1405163518

ISBN-13: 9781405163514

In case you prescribe for sufferers with arrhythmias, you'll want to maintain this priceless paperback shut handy. the second one version of this useful reference responds to alterations within the to be had medicines in addition to within the manner they're at present used.

The e-book studies every little thing you must comprehend and prescribe today’s antiarrhythmic drugs:

  • mechanisms of cardiac arrhythmias and the way antiarrhythmic medicines regulate these arrhythmias, together with universal antagonistic results
  • which elements to think about in utilizing those medicines for therapy of supraventricular tachyarrhythmias, ventricular arrhythmias, and arrhythmias in being pregnant
  • a exact evaluation of atrial traumatic inflammation that will help you make judgements for sufferer administration during this advanced area

Dr. Fogoros considers all of the most up-to-date medicinal drugs, plus promising medications less than research, to offer you an entire photo of healing ideas. With Antiarrhythmic medicines: a realistic Guide, moment version, you have liable info on how each one drug works and whilst every one is indicated so that you may give your sufferers the absolute best treatment.Content:
Chapter 1 Mechanisms of Cardiac Tachyarrhythmias (pages 1–35):
Chapter 2 advent to Antiarrhythmic medicines (pages 36–51):
Chapter three classification I Antiarrhythmic medications (pages 53–79):
Chapter four category II Antiarrhythmic medicinal drugs; Beta?Blocking brokers (pages 80–85):
Chapter five category III Antiarrhythmic medicinal drugs (pages 86–101):
Chapter 6 classification IV medications: Calcium?Blocking brokers (pages 102–106):
Chapter 7 Unclassified Antiarrhythmic brokers (pages 107–111):
Chapter eight Investigational Antiarrhythmic medicinal drugs (pages 112–116):
Chapter nine universal antagonistic occasions with Antiarrhythmic medicines (pages 117–129):
Chapter 10 uncomplicated ideas of utilizing Antiarrhythmic medicines (pages 131–137):
Chapter eleven remedy of Supraventricular Tachyarrhythmias (pages 138–150):
Chapter 12 therapy of Ventricular Arrhythmias (pages 151–163):
Chapter thirteen remedy of Arrhythmias in being pregnant (pages 164–167):

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Additional resources for Antiarrhythmic Drugs: A Practical Guide, Second Edition

Example text

The U wave is the ECG manifestation of the EAD itself. The T-U abnormalities tend to be dynamic; that is, they wax and wane from beat to beat, mainly depending on beatto-beat variations in heart rate. The slower the heart rate, the more exaggerated the T-U abnormality; hence, this condition is said to be pause dependent. 17. , a large EAD). The large EAD, in turn, produces another burst of tachycardia. Pause-dependent triggered activity should be strongly suspected whenever this ECG pattern is seen, especially in the setting of overt QT prolongation or in the setting of conditions that predispose to QT prolongation.

1 Vaughan-Williams classification system of antiarrhythmic drugs Class I: Sodium-channel-blocking drugs Class IA: Moderately slow conduction and moderately prolong action potential duration Quinidine Procainamide Disopyramide Class IB: Minimally slow conduction and shorten action potential duration Lidocaine Mexiletine Tocainide Phenytoin Class IC: Markedly slow conduction and minimally prolong action potential duration Flecainide Encainide Propafenone Moricizine* Class II: Beta-blocking drugs Class III: Prolong action potential duration Amiodarone Sotalol Ibutilide Dofetilide Class IV: Calcium-channel-blocking drugs ∗ Moricizine does not display the same affinity for the sodium channel as for other Class IC drugs, and some classify it as a IB drug.

They are discussed in more detail in Chapter 12. CHAPTER 2 Introduction to antiarrhythmic drugs All cardiac tachyarrhythmias—whether caused by abnormal automaticity, reentry, or channelopathies—are mediated by localized or generalized changes in the cardiac action potential. Thus, it should not be surprising that drugs that alter the action potential might have important effects on cardiac arrhythmias. How antiarrhythmic drugs work Thinking of an antiarrhythmic drug as a soothing balm that suppresses an “irritation of the heart” is more than merely naive; it is dangerous.

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Antiarrhythmic Drugs: A Practical Guide, Second Edition by Richard N. Fogoros(auth.)

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